There is no cure for amyotrophic lateral sclerosis (ALS), also known as Lou Gehrig's Disease, but new findings from the University of Missouri School of Medicine and the University of Missouri College of Veterinary Medicine ...
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Researchers from North Carolina State University have identified proteins that may be useful in both earlier diagnosis of Amyotrophic Lateral Sclerosis (ALS) and in more accurate disease prognosis.
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Promising findings from preclinical animal studies show the potential of gene therapy for treating incurable neurological disorders. In new research presented today, scientists successfully used gene therapy to slow the progression ...
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Promising new research by Christian Mueller, Ph.D., and Robert H. Brown Jr., DPhil, MD, at the University of Massachusetts Medical School, provides evidence that a therapy using synthetic microRNAs may safely treat patients ...
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Toxic protein assemblies, or "amyloids," long considered to be key drivers in many neuromuscular diseases, also play a beneficial role in the development of healthy muscle tissue, University of Colorado Boulder researchers ...
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Houston Methodist researchers have discovered a repair defect in nerve cells that send messages to the brain, opening the door to a possible therapy for preventing or slowing down ALS. Also known as Lou Gehrig's disease, ...
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Unlike old dogs, old adults can indeed learn new tricks thanks to a protein molecule called netrin.
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(HealthDay)—There's growing evidence that the herpes virus responsible for cold sores also may cause Alzheimer's disease, a new research paper contends.
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Virginia Tech Carilion Research Institute scientists have shown that mutations in specific genes that destroy motor neurons and thereby cause the devastating effects of amyotrophic lateral sclerosis— also known as ALS or ...
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A genetic defect tied to a variety of neurodegenerative diseases and mental illnesses changes how cells starved of sugar metabolize fatty compounds known as lipids, a new study led by researchers from the Johns Hopkins Bloomberg ...
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The normal function of a gene associated with the neurodegenerative diseases amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD) has been determined for the first time by University of Bath scientists.
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A mechanism for amyotrophic lateral sclerosis (ALS) development has been discovered at Umeå University, Sweden. The researchers report that proteins with a defect structure spread the deformation to other proteins. The discovery ...
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"Determining the mechanism that may be contributing to nerve cell damage could help us develop new drugs that better treat the disability caused by multiple sclerosis," said USask neurology professor Dr. Michael Levin, Saskatchewan ...
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A group of Massachusetts General Hospital (MGH) investigators is proposing that targeting immune checkpoints—molecules that regulate the activity of the immune system—in immune cells called microglia could reduce the ...
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Four out of five people with motor neuron disease are likely to experience changes in their brain function, as well as impaired movement, research suggests.
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Amyotrophic lateral sclerosis (ALS), also referred to as Lou Gehrig's disease in American English and Motor Neurone Disease in British English, is a form of Motor Neuron Disease caused by the degeneration of upper and lower neurons, located in the ventral horn of the spinal cord and the cortical neurons that provide their efferent input. The condition is often called Lou Gehrig's disease in North America, after the New York Yankees baseball player who was diagnosed with the disease in 1939. The disorder is characterized by rapidly progressive weakness, muscle atrophy and fasciculations, spasticity, dysarthria, dysphagia, and respiratory compromise. Sensory function generally is spared, as is autonomic and oculomotor activity. ALS is a progressive, fatal, neurodegenerative disease with most affected patients dying of respiratory compromise and pneumonia after 2 to 3 years; although some perish within a year from the onset of symptoms, and occasional individuals have a more indolent course and survive for many years.
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