A virulent group of tuberculosis germs spread from East Asia in waves propelled by industrialisation, World War I and Soviet collapse to yield some of the drug-resistant strains plaguing the world today, a study said Monday.
Jan 19, 2015
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The body responds to tuberculosis infection by locking the bacterial offenders into tiny clusters of immune cells called granulomas, which are a hallmark of the disease. This containment strategy succeeds at first, but eventually ...
Nov 24, 2014
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Using a high-throughput screening assay, EPFL scientists have discovered two small molecules that could overcome the multidrug resistance of the bacterium that causes tuberculosis.
Oct 8, 2014
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Over the past few years, a class of compounds called ADEPs (cyclic acyldepsipeptides) has emerged as a promising new weapon in the fight against drug-resistant bacteria. The compounds work by attaching themselves to a cellular ...
Sep 29, 2014
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UCLA-led study has identified a protein that appears to play a key role in protecting people infected with Mycobacterium tuberculosis—the bacterium that causes tuberculosis—from developing the active form of the disease. ...
Aug 20, 2014
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The lung lesions in an individual infected with tuberculosis (TB) are surprisingly variable and independent of each other, despite whether the patient has clinically active or latent disease, according to a new animal study ...
Dec 15, 2013
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A protein at the center of Parkinson's disease research now also has been found to play a key role in causing the destruction of bacteria that cause tuberculosis, according to scientists led by UC San Francisco microbiologist ...
Sep 4, 2013
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(Medical Xpress) -- New discoveries by Cambridge scientists about a molecular waste-disposal process that ‘eats’ bacteria are influencing the clinical management of cystic fibrosis, and could be the basis of innovative ...
Apr 4, 2012
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UCLA researchers pinpointed a new mechanism that potently activates T-cells, the group of white blood cells that play a major role in fighting infections.
Mar 25, 2012
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A team of UCLA scientists has found that the pathogen that causes leprosy has a remarkable ability to avoid the human immune system by inhibiting the antimicrobial responses important to our defenses.
Jan 29, 2012
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