Airborne particles beyond traffic fumes may affect asthma risk

Researchers in Sydney and Newcastle, Australia have found that elements of dust, particularly those coarse particles that contain iron traces, stimulate the production of inflammatory molecules in cells from the airways of mice and healthy human volunteers.

Surprisingly, traffic fume pollutants did not cause these changes.

The findings are featured in a new Respirology study.

"These effects are likely to contribute to the development of asthma in childhood, as well as to worsening of asthma when are high." said Dr. Rakesh Kumar, lead author of the study.

"Our findings emphasize that larger airborne particles derived from dust may have important adverse effects on ."

Explore further

First model of how Sydney pollutants may drive childhood asthma

More information: Kumar, R. K., Shadie, A. M., Bucknall, M. P., Rutlidge, H., Garthwaite, L., Herbert, C., Halliburton, B., Parsons, K. S. and Wark, P. A.B. (2014), Differential injurious effects of ambient and traffic-derived particulate matter on airway epithelial cells. Respirology. DOI: 10.1111/resp.12381
Journal information: Respirology

Provided by Wiley
Citation: Airborne particles beyond traffic fumes may affect asthma risk (2014, September 15) retrieved 30 March 2020 from
This document is subject to copyright. Apart from any fair dealing for the purpose of private study or research, no part may be reproduced without the written permission. The content is provided for information purposes only.

Feedback to editors

User comments